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鸡传染性喉气管炎是由疱疹病毒引起的一种急性传染病。本病多流行于晚秋、冬季和早春季节,各种日龄和品种的鸡都可感染此病,主要发生于2月龄以上的鸡,以5～8月龄者最易感。发病突然,且在鸡群内传播快、发病急,发病率达60％～95％,死亡率达10％-30％。现就一起鸡传染性喉气管炎的诊治情况介绍如下。 相似文献
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鸡传染性喉气管炎是由疱疹病毒引起的一种急性呼吸道传染病,严重的鸡群发病率接近100％,死亡率10％-40％不等,一般发病率为20％-50％,死亡率2％-5％。产蛋鸡表现为产蛋量明显下降,畸形蛋、沙皮蛋、软皮蛋增多。雏鸡也有发病,但发病率低,其死亡率不如产蛋鸡明显。 相似文献
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传染性喉气管炎是由鸡传染性喉气管炎病毒引起的鸡的一种急性上呼吸道接触性传染病，以传播快、呼吸困难、咳血痰等为特征，可造成鸡100％发病，死亡率为10％-40％，并且能严重影响鸡的产蛋量，对养鸡业造成一定的危害。该病无特效治疗药物，所以最好采取接种疫苗来预防。目前国内外用于防治本病的疫苗基本上都是弱毒疫苗。据报道，也有人研制使用灭活苗，但效果不理想。而目前常使用的弱毒疫苗存在以下缺点：毒力普遍偏强。 相似文献
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《中国家禽》2016,(8)
为了评估鸡传染性喉气管炎痘病毒载体重组基因工程疫苗威多妙喉痘(Vector-mune FP-LT)对鸡传染性喉气管炎的免疫效果,采用SPF鸡进行免疫攻毒保护试验。试验结果表明:胚源传染性喉气管炎病毒疫苗(CEO疫苗)和组织培养的传染性喉气管炎病毒疫苗(TCO疫苗)免疫后分别有50%和10%的鸡表现出免疫副反应,而威多妙喉痘免疫后无不良反应;CEO疫苗免疫组、TCO疫苗免疫组和威多妙喉痘免疫组的攻毒保护率分别为100%、90%、85%,无显著差异;气管病变计分结果与临床表现结果一致。同时进行的同居感染试验结果表明:非免疫同居感染组保护率仅为60%,而威多妙喉痘免疫后同居感染的保护率为100%。 相似文献
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鸡新城疫标准强毒感染鹅试验 总被引：11，自引：0，他引：11
用鸡新城疫中国标准强毒F48E8及国外标准强毒Hert33、AG68人工感染鹅，可以使鹅感染发病，F48E8攻毒组发病率100％，死亡率50％（9／18）；AG68、Hert33攻毒组发病率明显低于F48E8攻毒组和JS2／98／Goose攻毒组，死亡率分别为6％（1／15）和0％（0／15）。对照组健康，且扑杀时平均体重明显高于攻毒组。实验证明：新城疫中国标准强毒株F48E8等毒株可以使鹅发病，症状和病变都与文献中所报道的鹅副粘病毒病相同。 相似文献
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鸡传染性喉气管炎是由疱疹病毒引起的急性传染病。本病多流行于晚秋、冬季和早春季节,各种日龄和品种的鸡都可感染此病,主要发生于2月龄以上的鸡,以5-8月龄易感。发病突然,且在鸡群内传播快。病初,病鸡眼部流泪、红肿,流鼻水,呼吸有锣音。本病发病率达60％～95％,死亡率达10％-30％。所以对养鸡业造成很大危害。现就一起鸡传染性喉气管炎的诊治情况介绍如下。 相似文献
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<正>传染性喉气管炎是由疱疹病毒引起的鸡的急性呼吸道传染病,各种年龄的鸡均可感染,4～10月龄的成年鸡感染时出现明显的特征性症状。传染性喉气管炎的主要传染源是病鸡和康复带毒鸡。本病在鸡群中传播较快,感染发病率可达90%～100%,死亡率为5%～70%。传染性喉气管炎一年四季均可发生,多流行于秋、冬和春季。笔者在近年多次在6-7月诊治中发现鸡传染性喉气管炎。现报道如下: 相似文献
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The efficacy of four different commercial live vaccines (vaccines A, B, C, and D) against the infectious laryngotracheitis virus (ILTV) was assessed in specific-pathogen-free (SPF) chickens. SPF chickens were vaccinated intraocularly at 6 wk old with ILTV live vaccines and were challenged intratracheally with the N91B01 strain of virulent Korean ILTV 2 wk after vaccination. The immunity against ILTV live vaccines was assessed by the incidence of latent infection by the challenge virus in the chickens' tracheas and trigeminal ganglia, the reisolation rate of the challenge virus, and the clinical signs in the chickens challenged with the N91B01 strain of ILTV. The latent infection in chickens was assessed by nested polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP). Our data showed that the clinical signs and challenge virus isolation were negative in all chickens receiving four difference commercial ILTV live vaccines. The viral DNA of the vaccine strain, but not that of the challenge virus, was detected in chickens vaccinated with vaccine A by nested PCR-RFLP. The viral DNAs of both the vaccine and challenge strains were detected from chickens vaccinated with vaccines B, C, and D. This study showed that only vaccine A can protect chickens from latent infection with the field virulent ILTV. We speculate that the efficacy of infectious laryngotracheitis live vaccines to protect chickens from latent infection with virulent ILTVs can be assessed by nested PCR-RFLP analysis. 相似文献
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湖南鸡隐孢子虫感染情况调查及雏鸡人工感染试验 总被引：2，自引：0，他引：2
作者对湖南省５县（市）８个鸡场的３４８只鸡进行了隐孢子虫感染的调查，并用２５只雏鸡作了隐孢子虫的人工感染试验。调查表明：４个县（市）有鸡隐孢子虫感染，感染率为２．８～２５．０％，总感染率为１２．１％，其中２～４周龄鸡为３．６％，８～４０周龄鸡为１５．１％，伊莎鸡、ＡＡ鸡、罗曼鸡、艾维茵鸡、泰国肉鸡分别为２１．９％、１３．６％、９．１％、５．４％和０；１０只阳性鸡器官粘膜涂片检查盲肠、法氏囊、直肠的染虫率分别为１００％、６０％和４０％，气管、十二指肠和小肠中段未查到虫体。雏鸡人工感染试验表明：经鼻腔和眼结膜感染潜隐期均为５天；经口感染潜隐期为４天，第８～１９天排出卵囊数较多，第１２天排卵囊达到最高峰，显露期为２１天。 相似文献
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通过采用内服中药、中药散直接喷贴咽喉病灶处的内外齐攻的双联法治疗病鸡。人工发病试验结果表明,该方法有缓解和治疗鸡传染性喉气管炎作用,其中双联法组存活率比西药泰乐菌素注射液组高27．71％、比攻毒对照组高50．96％;双联法组的存活率明显高于其他组。存活率与攻毒不给药组比较,存活率统计学有显著性差异（P〈0．05）;和空白对照组比较,存活率差异不显著（P〉0．05）;且试验组与空白对照组体重无明显差异,说明该药不影响雏鸡的正常生长发育。临床试验结果也表明,治疗鸡喉气管炎效果显著,治愈率达95％以上。 相似文献
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“表达H5N1禽流感病毒HA和NA基因的重组鸡痘病毒（rFPV—AI）疫苗”以及“表达传染性喉气管炎病毒gB基因的重组鸡痘病毒（rFPV—ILT）疫苗”均已在实现商业化生产。由于两种疫苗使用了相同的载体病毒,如何使用才能减少相互干扰而产生最好的免疫效果。本研究设计了三个试验组：1）免疫rFPV—AI后间隔4周接种rFPV—ILT;2）rFPV—AI和rFPV—ILT混合后接种;3）将rFPV—AI和rFPV—ILT分别在两个翅膀同时免疫。结果表明,试验鸡接种rFPV—AI后4周接种rFPV—ILT,对传染性喉气管炎病毒WG株攻击的保护率为60％（6／10）,低于rFPV—ILT单独免疫组的保护率（100％,10／10）;rFPV—AI和rFPV—ILT混合后免疫组对禽流感病毒强毒攻击的保护率为80％（8／10）,对传染性喉气管炎病毒强毒攻击的保护率为70％（7／10）,而rFPV—AI和rFPV—ILT分两点同时接种对两种病毒攻击均能产生完全保护。本试验结果建议将这两种相同载体的重组病毒疫苗分两点同时接种以避免相互之间的干扰。 相似文献
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Experiments were undertaken to study the pathogenesis of VRI-33, a strain of fowl adenovirus serotype 8 isolated from the liver of a broiler chicken with inclusion body hepatitis. A 30% death rate resulted from oral infection of one-day-old specific pathogen free chickens with 106 plaque forming units of VRI-33. Chickens 10, 14, 21 and 28 days of age did not die following infection via natural routes but there were some motalities following infection via parenteral routes. Immunodepression by neonatal cyclophosphamide treatment, followed by infection with VRI-33 via non-parenteral routes, caused varying degrees of hepatitis with basophilic intranuclear inclusion bodies in hepatocytes. The mortality rate of cyclophosphamide-treated, VRI-33 infected chickens, was not significantly altered by post-infection temperature stress. Infection with infectious bursal disease virus, followed by infection with VRI-33 via natural routes at 14 days of age, was not associated with mortalities. 相似文献
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几种病毒与禽病原性大肠杆菌的人工联合感染 总被引：6，自引：0，他引：6
以2种剂量的低致病性禽流感病毒(lowly pathogenic avian influenza virus,LPAIV)，传染性支气管炎病毒（infectious bronchitis virus,IBV)疫苗株H120和H52，新城疫病毒（Newcastle disese viurs,NDV)Lasota株分别于气管内注射10日龄易感鸡，2d后，气管注射禽病原性大肠杆菌O37株（O78），连续观察5d，结果，除LPAIV单独感染组有6．25％的死亡率外，其余各病毒单独接种组均健活；大肠杆菌O37株单独接种组的死亡率为62．50％，较高剂量的LPAIV，IBV H120和H52，NDV Lasota株与大肠杆菌O37株有效强的协同致病作用，死亡率分别达到81.25%,100.00%,93.75%和87.50%，而较低剂量的上述病毒则无明显的协同作用，IBV，NDV疫苗株与大肠杆菌联合接种组的多数死亡鸡病程推迟。 相似文献
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O. L. Nielsen J. C. Jensenius P. H. Jrgensen S. B. Laursen 《Veterinary immunology and immunopathology》1999,70(3-4):309-316
Mannan-binding lectin (MBL) is a serum collectin which is believed to be an opsonin of the innate immune defence against various microorganisms. MBL is a minor acute phase reactant in man. We investigated the concentration of serum MBL in chickens infected with infectious bronchitis virus (IBV) and infectious laryngotracheitis virus (ILTV). The concentration of serum MBL increased about twofold (from approximately 6 to 12 μg/ml) due to these viral infections. The concentration peaked 3–7 days after infection with IBV, and 3–5 days after ILTV infection, depending on the ILTV strain used. The increased levels returned to normal values 6–10 days after infection. The results indicated that MBL is a minor acute phase reactant in chickens. 相似文献
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表达传染性喉气管炎病毒gB基因重组鸡痘病毒疫苗免疫持续期试验 总被引：2，自引：0，他引：2
将200003批疫苗免疫4周龄SPF鸡,免疫后的7、14、21、30、60、90、120、150、180和225 d分别采血,分离血清,采用ELISA方法检测血清抗FPV抗体,结果表明重组疫苗免疫后14 d,免疫鸡血清抗体已经全部阳转,免疫后的21 d血清抗FPV的抗体出现峰值,此后便开始下降,到免疫后的6个月抗体水平已经接近阴性对照的水平.用ILTV WG株和FPV 102株强毒进行的攻毒保护试验与血清检测的结果基本一致,在免疫后的5个月内可以使免疫鸡获得100%(10/10)保护,免疫后的6个月对ILT和FP的免疫保护分别为1/7和2/10,此时需要对鸡群实施二次免疫.其他5批疫苗(200001、200002、200101、200102和200103)免疫SPF鸡后5个月用ILTV WG株和FPV 102株攻击也获得了完全保护. 相似文献
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Specific-pathogen-free chickens were infected via the trachea when 4 weeks old with 2000 plaque-forming units (PFU) of the virulent Australian infectious laryngotracheitis (ILT) virus strain CSW-1. Titers of ILT virus in the trachea were greatest (10(7.0) PFU/ml in washings, 10(6.0) PFU/g of tissue) 2-4 days postinfection (PI). Infectivity then declined rapidly, to become undetectable by 7 days PI, although highly localized areas of ILT antigen in the tracheal epithelium were occasionally observed by fluorescent antibody staining at 7 and 8 days PI. Tracheal organ cultures established 7 and 8 days PI provided no evidence of latent ILT virus infection at this immediate post-acute stage of pathogenesis. ILT virus was not isolated from peripheral blood leukocytes or lymphoid organs (spleen, bursa, thymus). ILT virus was found in the trigeminal ganglia and/or brain in 14 of 36 chickens (40%) examined between 4 and 7 days after intratracheal inoculation, but it was not in these tissues in five chickens examined at 8 days PI. Virus was also detected at 6 days PI in the trigeminal ganglia in one of five chickens infected by the conjunctival route. These data indicate that the early pathogenesis of ILT (CSW-1) infection frequently involves the tissues of the nervous system. In acute ILT in 4-week-old chickens, interferon-alpha/beta activity was not detectable in serum or tracheal exudates within 14 days PI, but tracheal washings contained significant virus-neutralizing activity by 7 and 8 days PI. In 3-day-old chickens infected via the trachea with 200 PFU of ILT CSW-1, the clearance of ILT virus from the trachea was similar to that observed in 4-week-old chickens, but ILT virus spread systemically to the livers of 20% by 5-7 days PI. 相似文献