共查询到19条相似文献，搜索用时 812 毫秒
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羊口疮是由于羊的口疮病毒发生感染所导致,传播性极强,也就是俗称的羊传染性脓疱病,不仅只是在牲畜中进行传播,还会造成人的疾病感染因此必须及时做好羊口疮病的诊断与防治。本文将主要围绕着羊口疮病的诊断与防治对策进行讨论分析。 相似文献
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羊传染性脓疱病也称为羊传染性脓疱口炎、羊传染性脓疱皮炎,俗称为羊口疮,是由于感染羊传染性脓疱病毒而发生的一种人畜共患传染病,主要通过接触传播,在全世界任何养羊国家都能够发生。该病主要特征是唇、口腔黏膜以及局部皮肤出现红疹、脓疱、溃疡、结痂,由于通常在口唇附近发生病变,对采食饲草、饲料和吮吸乳汁产生影响,从而导致增重缓慢。如果对该病没有及时采取治疗,会由于机体衰竭或者继发引起肺炎等疾病而导致死亡。 相似文献
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羊传染性脓痂皮炎又被称为羊传染性脓疱病、羊口疮病,是由羊传染性脓疱病毒感染引发的一种急性接触性传染性疾病。羊养殖主产区均有发生,是一种常见疾病。羊传染性脓痂皮炎虽然造成的死亡率较低,但是会严重影响羊群正常采食,造成营养物质供给不足,身体逐渐消瘦,抵抗能力变差,易继发感染多种传染性疾病,表现出复杂的临床症状,威胁羊群健康。 相似文献
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秦玉中 《畜牧兽医科学(电子版)》2019,(3):118-119
羊传染性脓疱病是由传染性脓疱病毒感染引起的一种高度接触性、传染性疾病,对羊的健康生长造成的危害十分严重,常呈群发流行。临床上主要表现为口唇等皮肤和粘膜形成丘疹、脓疱、溃疡,最终形成疣状结痂。羊传染性脓疱病的发生与饲养管理有密切联系。在日常粗饲料投喂中,饲料切割长度不合理,饲料较硬,导致口腔黏膜受损,易为该种病毒传播流行提供条件。 相似文献
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羊口疮又被称为传染性脓疱病,引发该病的主要原因是病毒,会在肉羊之间传染。在传染性脓疱病发作时,病羊的鼻子、口腔、乳房等部位会生出脓疱、丘疹、水疱等,这也是肉羊传染性脓疱发作的主要症状表现。一旦肉羊传染性脓疱病暴发,会造成大面积感染,甚至还会造成羊群死亡。养殖户应注重肉羊传染性脓疱病的诊治,结合实际发病程度展开有效的处置措施。主要论述羊传染性脓疱病诊断与中医防控。 相似文献
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正羊传染性脓疱是由传染性脓疱病毒引起的山羊的传染性疾病,俗称"羊口疮"。该病的特征是患羊唇部皮肤黏膜形成丘疹、脓疱、溃疡和厚痂,重症病羊采食困难,逐渐消瘦,部分病羊继发其他疾病而死亡。1流行病学羊传染性脓疱病的病原体是羊口疮病毒,属于痘病毒属。该病毒在上皮细胞浆内繁殖,其所有毒株的抗 相似文献
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赵昌松 《畜牧兽医科学(电子版)》2021,(5):81-82
我国山区和平原面积辽阔,具有丰富的草资源,为养羊业发展提供源源不断的天然物质基础。随着我国羊肉消费能力的增长,到2019年全年羊肉产量达488万t,增长2.6%,许多养殖户更加重视羊群健康和疾病防治。羊传染性胸膜肺炎是一种高接触性传染病,具有传播速度快、致病性强和死亡率高等特点,给养殖户造成较大的经济损失。该文对羊传染性胸膜肺炎的发病原因、流行特点、临床症状、病理变化、诊断方法和防治措施进行论述。 相似文献
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羊传染性脓疱病研究进展 《畜牧与饲料科学》2022,43(3):117-121
羊传染性脓疱病是由羊传染性脓疱病毒（orf virus,ORFV）引起的传染性极强且流行广泛的人畜共患病。健康羊只感染ORFV后,会在唇、鼻和口腔等无毛部位依次形成丘疹、水疱、脓疱及溃疡,最后形成疣状结痂,产生增生性损伤。羔羊由于免疫系统发育不完全尤其易感,发病后会导致采食困难、营养物质摄入不足,进而影响生长发育,严重者会因继发性感染而死亡。结合近年来国内外研究报道,从病原学、流行病学、临床症状、诊断方法、预防和治疗措施等方面对羊传染性脓疱病进行综述,以期为兽医临床中该病的预防、诊断和治疗提供参考。 相似文献
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The epidemic of bovine spongiform encephalopathy (BSE) has been the most expensive disaster ever to have befallen farming in the UK. It is believed to have led to a new form of spongiform encephalopathy in humans and as yet there is no way of knowing how many people will die of this disease. In order to curtail the BSE epidemic major decisions had to be made, often on the basis of inadequate scientific data. These data may have been derived from experiments using small sample numbers. Here we review some examples of where this has happened, sometimes with a beneficial outcome and sometimes with a misleading outcome. The identification of BSE as a new disease depended on precise neuropathological observation of a small number of cases rather than the obvious occurrence of large numbers of sick animals. Similarly, the recognition that BSE may have led to disease in humans was based on the neuropathological and clinical picture of new variant Creutzfeldt-Jakob disease (CJD) rather than on an increase in the number of cases of CJD in the UK. Early in the BSE epidemic the possibility that disease could be maternally transmitted from cow to calf was raised, mainly because of a belief that such transmission occurs in scrapie disease of sheep. But, we argue, the evidence for maternal transmission of scrapie, collected in the 1960s, was based on small numbers and is inadequate. Subsequent research has shown a very substantial genetic component in scrapie and epidemiological data show no excess risk in the offspring of affected ewes relative to the risk in the offspring of affected rams. An experiment to determine whether maternal transmission occurs in BSE was flawed and was unable to distinguish between maternal transmission and genetic susceptibility to environmental contamination. An assessment of the risk of BSE to humans depends on determining the levels of infectivity in tissues and transmissibility across species. Data on both of these are deficient, so it is not possible to predict how many people in the UK or elsewhere will become affected with new variant CJD in the next fifty years. The assessment of whether BSE could be transmitted to sheep and whether sheep therefore pose a risk to humans is hampered by a serious lack of evidence about the epidemiology of scrapie in the UK and elsewhere. The UK has paid a heavy price for the BSE epidemic but lessons should be learned from the experience. Every country should have a Specified Offals Ban even if it has no cases of BSE because, by the time it has, it will be too late. Furthermore, the occasional case of BSE should not be regarded as insignificant since it may be the harbinger of an epidemic in the making. 相似文献
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Summary The epidemic of bovine spongiform encephalopathy (BSE) has been the most expensive disaster ever to have befallen farming in the UK. It is believed to have led to a new form of spongiform encephalopathy in humans and as yet there is no way of knowing how many people will die of this disease. In order to curtail the BSE epidemic major decisions had to be made, often on the basis of inadequate scientific data. These data may have been derived from experiments using small sample numbers. Here we review some examples of where this has happened, sometimes with a beneficial outcome and sometimes with a misleading outcome. The identification of BSE as a new disease depended on precise neuropathological observation of a small number of cases rather than the obvious occurrence of large numbers of sick animals. Similarly, the recognition that BSE may have led to disease in humans was based on the neuropathological and clinical picture of new variant Creutzfeldt‐Jakob disease (CJD) rather than on an increase in the number of cases of CJD in the UK. Early in the BSE epidemic the possibility that disease could be maternally transmitted from cow to calf was raised, mainly because of a belief that such transmission occurs in scrapie disease of sheep. But, we argue, the evidence for maternal transmission of scrapie, collected in the 1960s, was based on small numbers and is inadequate. Subsequent research has shown a very substantial genetic component in scrapie and epidemiological data show no excess risk in the offspring of affected ewes relative to the risk in the offspring of affected rams. An experiment to determine whether maternal transmission occurs in BSE was flawed and was unable to distinguish between maternal transmission and genetic susceptibility to environmental contamination. An assessment of the risk of BSE to humans depends on determining the levels of infectivity in tissues and transmissibility across species. Data on both of these are deficient, so it is not possible to predict how many people in the UK or elsewhere will become affected with new variant CJD in the next fifty years. The assessment of whether BSE could be transmitted to sheep and whether sheep therefore pose a risk to humans is hampered by a serious lack of evidence about the epidemiology of scrapie in the UK and elsewhere. The UK has paid a heavy price for the BSE epidemic but lessons should be learned from the experience. Every country should have a Specified Offals Ban even if it has no cases of BSE because, by the time it has, it will be too late. Furthermore, the occasional case of BSE should not be regarded as insignificant since it may be the harbinger of an epidemic in the making. 相似文献
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Megersa B Biffa D Abunna F Regassa A Bohlin J Skjerve E 《Tropical animal health and production》2012,44(7):1643-1651
A highly acute and contagious camel disease, an epidemic wave of unknown etiology, referred to here as camel sudden death syndrome, has plagued camel population in countries in the Horn of Africa. To better understand its epidemic patterns and transmission dynamics, we used epidemiologic parameters and differential equation deterministic modeling (SEIR/D-model) to predict the outcome likelihood following an exposure of susceptible camel population. Our results showed 45.7, 17.6, and 38.6 % overall morbidity, mortality, and case fatality rates of the epidemic, respectively. Pregnant camels had the highest mortality and case fatality rates, followed by breeding males, and lactating females, implying serious socioeconomic consequences. Disease dynamics appeared to be linked to livestock trade route and animal movements. The epidemic exhibited a strong basic reproductive number (R (0)) with an average of 16 camels infected by one infectious case during the entire infectious period. The epidemic curve suggested that the critical moment of the disease development is approximately between 30 and 40?days, where both infected/exposed and infectious camels are at their highest numbers. The lag between infected/infectious curves indicates a time-shift of approximately 3-5?days from when a camel is infected and until it becomes infectious. According to this predictive model, of all animals exposed to the infection, 66.8 % (n?=?868) and 33.2 % (n?=?431) had recovered and died, respectively, at the end of epidemic period. Hence, if early measures are not taken, such an epidemic could cause a much more devastative effect, within short period of time than the anticipated proportion. 相似文献
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von Tavel L Fivian R Kirchhofer M Boujon P Hirsbrunner G 《Schweizer Archiv für Tierheilkunde》2005,147(10):445-452
In spring 2005, the outbreak of contagious abortion caused by Salmonella Abortusovis in 6 sheep flocks in Switzerland led to considerable economic losses. The Swiss small ruminant health service (BGK) evaluated this case. The aim was to identify the source of the epidemic in order to avoid further spread of infection and to evaluate the possibility of using vaccination. Moreover, a strategy for prevention of future outbreaks was developed. This article aims to increase disease awareness of food animal practitioners for Salmonella Abortusovis abortion in sheep. 相似文献