共查询到18条相似文献，搜索用时 203 毫秒
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库拉索芦荟是芦荟大家族中应用最广泛的一个品种,它栽培容易,养护简单,非常适合家庭室内观赏种植。阐述库拉索芦荟的生物学特征,重点介绍其室内盆栽的繁殖、栽培、管理技术,同时还介绍了它在生活中的应用。 相似文献
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芦荟 ,又名油葱、龙角、狼牙掌 ,系百合科芦荟属多年生肉质草本。原产热带非洲 ,唐代中叶传入我国。品种有几百个 ,但具有营养和药用价值的只有几种 ,如好望角芦荟、树芦荟、库拉索芦荟、元江芦荟、中华芦荟、皂质芦荟等。其肉质叶片由基部生出 ,簇生 ,呈尖舌状 ,叶两边有刺状小齿 ,四季青翠 ,生意盎然。到了冬季和早春叶丛中抽出花多为深红、橙黄或黄色、偶有白色。早在一二千年以前被古埃及人誉为“万应灵药”。 芦荟含有大量植物蛋白 ,1 2种维生素 ,叶绿素 ,芦荟素 ,芦荟大黄素 ,胆碱 ,配糖体 ,多种微量元素 ,活性酶类和 2 2种氨基酸 … 相似文献
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以不同年代、不同品种的芦荟为试验材料,采用蒽酮-硫酸法对芦荟多糖进行研究,并研究了Folin-酚A、Folin-酚B对芦荟蛋白质含量以及水分的影响。结果表明:生长期对多糖得率的影响显著,6年生芦荟显著优于其它年份,中华芦荟多糖含量显著优于库拉索芦荟,高达3.092mg/mL;6年生库拉索芦荟全叶中的蛋白质含量显著优于其它年份和品种,高达0.845mg/mL;6年生芦荟全叶及凝胶中水分含量显著低于其它年份,达97.70%,是药物病理等科研项目较为理想的研究对象,为开发医药、美容、保健等提供优良的原材料。 相似文献
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芦荟的栽培管理与应用前景 总被引：2，自引：0，他引：2
种植芦荟是上海郊区农业结构调整中新发展起来的种植业之一 ,发展速度较快 ,并逐渐走向规模化、系列化 ,今后必然会发展成为农、工、商、贸一体化的大型产业。上海市农业科学院园艺所于1974年从广东引进中华芦荟 ,作为中草药及观赏花卉种植。20世纪末上海市动植物引种中心引进菜用芦荟品种推广种植。近年来 ,随着对芦荟研究的深入和市郊农村产业结构的调整 ,不少单位开展了芦荟引种研究 ,目前本市引进的芦荟品种主要有 :(1)美国库拉索芦荟AloebarbadensisMill;(2)木立芦荟AloearberescensMill;(3)中华芦荟AloeveraL.varchinensis(Haw.)B… 相似文献
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芦荟是集医疗、美容、保健、观赏于一身的神奇植物,它含有70多种对人体有益的物质,具有健胃、通便、消炎、抗菌、抗癌等药理作用。目前芦荟已应用于医药、食品、美容、畜牧等工农业领域,是一种很有种植价值和开发前景的经济植物。笔者认为开发芦荟产品应从种植业入手,鼓励和引导农民种植高产质优的芦荟品种,如:美国库拉索芦荟(翠叶芦荟)、日本木立芦荟、中华芦荟、上农大叶芦荟等。另外,还要充分利用组织培养技术来发展芦荟种苗产业,大规模生产以提供大量优质种苗。发展芦荟规模种植的同时,还必须考虑产品的收购和加工,特别是粗加工,因为只… 相似文献
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塑料大棚栽培对库拉索芦荟蒽醌类物质的影响 总被引：1，自引：0，他引：1
以库拉索芦荟为材料,运用高效液相色谱技术,研究了塑料大棚栽培对库拉索芦荟主要有效成分蒽醌类物质的影响.结果表明:在大棚内生长的芦荟成熟叶总蒽醌类物质和芦荟素含量减少,同时,其高效液相色谱图中出峰数量也显著减少.该试验还研究了增强UV-B辐射可对芦荟有效成分的影响,试验证明,增强UV-B辐射促进芦荟叶中总蒽醌类物质和芦荟素的积累,在处理10 d内以每天照射6 h,总蒽醌和芦荟素含量增加最多. 相似文献
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AIM: To study the changes of the sphingomylinase activity and ceramide content in rabbit aorta of experimental atherosclerosis and investigate the effects of emodin on them. METHODS: The qualified rabbits were fed with food containing 1% cholesterol and 5% lard for 10 weeks to establish the animal models. The concentration of cholesterol (TC) was assayed by a enzyme method. Trace-fast-test method was used to test the activity of superoxide dismutase (SOD) and motified- BAMuGuoFu methods was employed to assay the content of myocardial malondialdehyde (MDA). Radiolabeled -enzyme-tracing was used to detect the activity of the sphingomyelinase,and thin-layered scanning was conducted to analyze the content of the ceramide in aorta. RESULTS: The ceramide content in aorta and the sphingomyelinase activity were markedly increased in the rabbits with experimental atherosclerosis. The increase was positively correlated with the content of TC and MDA and negatively correlated with the activity of SOD in blood. Compared to the model animals, emodin at concentration of 5 mg·kg-1, 10 mg·kg-1 and 20 mg·kg-1 respectively reduced the area of plague on endothelium in rabbit's aortic artery and elevated the activity of SOD (P<0.05). The activity of sphingomylinase and the content of ceramide were decreased at the same time (P<0.05). 10 mg·kg-1 emodin proved to be more effective than 5 mg·kg-1 and 20 mg·kg-1 emodin (P<0.01). CONCLUSIONS: Our data suggest that atherosclerosis is related to ceramide signal transduction initiated by factors such as oxidative stress in hypercholesterolemia. The emodin prevents the development of atherosclerosis probably by interfering with the above pathway. 相似文献
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WANG Shu-qiu LI Xiao-jie JIANG Zhi-mei QIN Xiao-yu HAN Yu-ze LIU Lei LIU Jun-xing WANG Fang-fang LIANG Yan-feng WANG Shu-xiang 《园艺学报》2011,27(6):1053-1058
AIM: To investigate the effects of Ganoderma lucidum spores on superoxide dismutase(SOD),malondialdehyde(MDA),total antioxidative capacity(T-AOC), cytochrome C, heat-shock protein 70 (HSP70), mitochondrial Ca2+ and brain-derived neurotrophic factor(BDNF) in the brain tissues of epilepsy rats.METHODS: The rat chronic epilepsy model was by intraperitoneal injection of pentetrazole(PTZ) at a subconvulsant dose (32 mg/kg).Flame atomic absorption method was used to detect the content of mitochondrial Ca2+,and spectrophotometer colorimetry was used to measure SOD activity,MDA content,T-AOC and cytochrome C levels in rat brain tissues. HSP70 and BDNF were determined by immunohistochemical method.RESULTS: The contents of mitochondrial Ca2+ and cytochrome C were higher, and the content of intracytoplasmic cytochrome C in the rat brain tissues was obviously lower in Ganoderma lucidum spores group than that in epileptic model group. Compared to epileptic model group, the activity of SOD and T-AOC in cytoplasm of the rat brain tissues decreased while MDA increased, and the numbers of BDNF-positive cells in cerebral cortex and hippocampus were significantly increased in Ganoderma lucidum spores group. The positive neuron population of HSP70 in hippocampus, basal nucleus and cortex was significantly higher in Ganoderma lucidum spores group than that in epilepsy model group.CONCLUSION: Ganoderma lucidum spores attenuate the impairment of neuronal mitochondria induced by seizure, and accelerate the expression of BDNF, resulting in restoring the energy metabolism in mitochondrion, thus alleviating the impairment and apoptosis of the brain tissues. 相似文献
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以金针菇液体发酵产菌丝为研究材料,用聚丙烯酰胺梯度凝胶电泳及抑制剂处理法,研究超氧化物歧化酶（SOD）的产生方式及其同工酶类型.结果表明,在液体培养过程中,SOD属于胞内酶,仅存在于菌丝中,其活性可被氯仿:乙醇和SDS抑制,但对KCN和H_2O_2不敏感,因此认为金针菇菌丝中仅以Mn-SOD为主。 相似文献
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AIM: To study the stability of mouse cerebral ischemia/reperfusion model induced by the method of monofilament. METHODS: Sixty male Kunming mice were divided into 3 groups according to the body weight: group A (18-21 g), group B (22-28 g) and group C (30-35 g). Ischemia/reperfusion (I/R) model was made by middle cerebral artery occlusion (MCAO) with nylon monofilament. To evaluate the mouse MCAO model, the method of PRM2 laser Doppler was used to detect the cerebral blood flow, the neurological deficit scores were determined by Longa standard and infarction volume was detected with TTC staining. The content of malondialdehyde (MDA) and activity of superoxide dismutase (SOD) were also measured by ELISA. RESULTS: The successful rates of model establishment in both group A and group B were higher than that in group C (P＜0.05), especially the highest in group B . The mortality in group A was significantly higher than that in group B and group C (P＜0.05). The behavior scores and cerebral infarct volume in group A and group B were significantly higher than those in group C (P＜0.05). Obvious brain injury and neurological deficits were also observed in group A and group B with the higher content of MDA and the lower activity of SOD in the cerebral cortex of the injury side. CONCLUSION: There are three important factors to ensure the success and stability of MCAO mouse model induced by monofilament, i.e. the diameter of monofilament matching the body weight of the mice, the suitable length of monofilament within the blood vessel, as well as the maintaining of proper room temperature during experiment. The MDA content and SOD activity are also effective indexes for evaluating the cerebral I/R injury. 相似文献
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以香蕉不定芽为材料,MS+6-BA 4.0 mg·L-1+NAA 0.2 mg·L-1为基本培养基,采用单因素试验设计方法,研究油菜素内酯对香蕉不定芽增殖及酶活性变化的影响。结果表明,随着香蕉不定芽的形成,不加油菜素内酯,SOD活性逐渐下降,然后急剧上升;POD活性先上升,然后不断下降。油菜素内酯处理下SOD活性总体呈下降-上升-下降的变化规律,POD活性呈下降-上升-下降的规律。培养基中加入0.3 mg·L-1油菜素内酯的香蕉不定芽中的可溶性糖(3.73 mg·g-1)、脯氨酸含量(67.10μg·g-1)和株高(5.00 cm)都显著高于其他处理。 相似文献
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AIM: To explore the role of NADPH oxidase 1 (NOX1) in tumor necrosis factor-α (TNF-α)-induced oxidative damage and inflammation in alveolar epithelial cells.METHODS: The mRNA and protein expression levels of NOX1 in alveolar epithelial cells after TNF-α treatment were determined by real-time PCR and Western blot. NOX1 siRNA and its negative control were transfected into the alveolar epithelial cells. After the induction of TNF-α, NOX1 levels in the cells were measured by real-time PCR and Western blot, and the content of malondialdehyde (MDA) in the cells was detected by thiobarbituric acid method. Xanthine oxidation assay was used to detect the activity of superoxide dismutase (SOD) in the cells. The contents of interleukin-4 (IL-4), IL-6 and IL-1β in cell culture medium were examined by ELISA. The rate of apoptosis was analyzed by flow cytometry. Western blot was used to detect the level of apoptotic protein cleaved caspase-3.RESULTS: The expression of NOX1 at mRNA and protein levels in TNF-α-induced cells was increased after induction (P<0.05). After transfection of NOX1 siRNA, the expression of NOX1 at mRNA and protein levels in the cell was downregulated (P<0.05). Transfection of siRNA negative control had no effect on the expression level of NOX1 in the cells. The content of MDA in the cells after TNF-α treatment was increased, the activity of SOD was reduced, the releases of IL-4, IL-6 and IL-1β by the cells were increased, and the apoptotic rate and the level of apoptotic protein cleaved caspase-3 were increased as compared with the cells that were not treated with TNF-α (P<0.05). The content of MDA in the cells with NOX1 knockdown induced by TNF-α was reduced, the activity of SOD elevated, and the releases IL-4, IL-6 and IL-1β, the apoptotic rate and the level of apoptotic protein cleaved caspase-3 decreased, as compared with the cells only treated with TNF-α induction (P<0.05).CONCLUSION: TNF-α induces the expression of NOX1 in the alveolar epithelial cells. Knockdown of NOX1 expression reduces cellular oxidative damage, releases of inflammatory factors, and cell apoptosis. 相似文献
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XIE Long-chang ZHANG Bo GAO Qing-chun FU Xian LI You-fu LIU Hong-ying WEI Huan 《园艺学报》2014,30(2):339-346
AIM:To investigate the mechanism in which DL-3-n-butylphthalide (NBP) protects the mitochondria from the damage of oxygen-glucose deprivation (OGD). METHODS:Human umbilical vein endothelial cells (HUVECs) were exposed to OGD to induce endothelial damage. Mitochondrial morphology and mitochondrial reactive oxygen species (ROS) were examined using MitoTracker Green and MitoSOX Red, respectively. The activity of superoxide dismutase (SOD) was evaluated by SOD assay kit. RESULTS:NBP significantly attenuated OGD-induced mitochondrial fragmentation, reduced the content of mitochondrial ROS and increased the activity of SOD. CONCLUSION:NBP alleviates OGD-induced damage in the mitochondria. Reduction of mitochondrial ROS and enhancement of SOD activity may be the mechanism in which NBP protects mitochondria. 相似文献
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AIM To investigate the effect of scutellarin （SCU） on oxidative stress and apoptosis induced by lipopolysaccharide （LPS） in human glomerular epithelial cells and its mechanism. METHODS Human glomerular epithelial cells were cultured in vitro ， and were treated with LPS （1.0 mg/L） to establish a cell injury model. The cells were divided into normal control （NC） group， LPS group， NC+SCU group， LPS+SCU group， LPS+miR-NC group， LPS+microRNA-7-5p （miR-7-5p） group， LPS+SCU+anti-miR-NC group and LPS+SCU+anti-miR-7-5p group. Cell viability was detected by CCK-8 assay. Apoptosis was detected by flow cytometry. The intracellular malondialdehyde （MDA） content and superoxide dismutase （SOD） activity， and lactate dehydrogenase （LDH） activity in the cell culture supernatant were determined by kit. RT-qPCR was used to detect the expression level of miR-7-5p. RESULTS Compared with NC group， the cell viability， miR-7-5p expression and SOD activity in LPS group were significantly reduced， and the apoptotic rate， MDA content and LDH activity were significantly increased （P <0.05）. Compared with LPS group， the cell viability， miR-7-5p expression and SOD activity in LPS+SCU group were significantly increased， and the apoptotic rate， MDA content and LDH activity were significantly reduced （P <0.05）. Compared with LPS+miR-NC group， the cell viability and SOD activity in LPS+miR-7-5p group were significantly increased， and the apoptotic rate， MDA content and LDH activity were significantly reduced （P <0.05）. Compared with LPS+SCU+anti-miR-NC group， the cell viability and SOD activity in LPS+SCU+anti-miR-7-5p group were significantly reduced， and the apoptotic rate， MDA content and LDH activity were significantly increased （P <0.05）. CONCLUSION Scutellarin inhibits LPS-induced oxidative stress damage and apoptosis in glomerular epithelial cells via up-regulating miR-7-5p expression. 相似文献