Ultrastructure of lymphatic cells in ovine leucosis     

R Rudolph  J Paulsen 《Berliner und Münchener tier?rztliche Wochenschrift》1972,85(5):85-87

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Bile acid fractionations by high-performance liquid chromatography in equine liver disease     

J. J. Kaneko  W. G. Rudolph  D. W. Wilson  G. P. Carlson 《Veterinary research communications》1992,16(3):161-172

Serum bile acids were fractionated by high-performance liquid chromatography (HPLC) in 13 control and 8 cases of liver disease in horses. The severity and type of liver injury was determined by histopathological examination of biopsy and/or necropsy specimens. The total serum bile acids (tSBA) were determined in these horses by an enzymatic method (SBA-EA) and by summation of the bile acids (SBA-LC) as fractionated by the HPLC. The SBA-LC were generally higher than the SBA-EA in both the controls and liver disease and they did not parallel each other. The primary bile acids, total cholates and total chenodeoxycholates accounted for most of the tSBA increases in liver disease. There was a shift in profile from taurocholate to free (unconjugated) cholate in direct relation to the severity of the liver injury. Among the secondary bile acids, total deoxycholates and total taurodeoxycholates increased at random. The pattern of the SBA profile in relation to the severity of the liver disease suggested that hepatocellular excretion is the most sensitive step in the enterohepatic circulation of the bile acids.  相似文献   

Specificity of the booster effect     

K W Perlberg  W Rudolph 《Archiv fuer experimentelle veterinaermedizin》1972,26(1):47-56

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Classification of leucoses and reticuloses     

R Rudolph 《Berliner und Münchener tier?rztliche Wochenschrift》1973,86(22):432-434

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C-Typ Viruspartikel in Blutlymphozytenkulturen von zwei Schafen mit persistierender Lymphozytose     

E. Weiss  J. Paulsen  R. Rudolph  R. Hoffmann 《Zoonoses and public health》1971,18(3):244-248

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Equine piroplasmosis: leukocytic response to Babesia equi (Laveran, 1901) infection in Chile.     

W Rudolph  J Correa  L Zurita  W Manley 《The British veterinary journal》1975,131(5):601-609

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On the histopathology of skin changes in the experimental erysipelas infection of pigs. I. Skin changes at the scarification incision in infection based on the Fortner and Dinter method     

W Rudolph 《Archiv fuer experimentelle veterinaermedizin》1967,21(3):629-644

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Experimental transmission of lymphatic leucosis of sheep]     

J Paulsen  R Rudolph  T Schliesser 《Zentralblatt für Veterin?rmedizin. Reihe B. Journal of veterinary medicine. Series B》1975,22(9):737-748

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Bovine leukosis: a comparative study of three haematological diagnostic keys in Chilean cattle     

W Rudolph  W Cripe  D Hird  K Rusch  A Soffia  G Villouta 《The British veterinary journal》1972,128(10):506-511

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The Functional Role of Oxytocin in the Induction of Oocyte Meiotic Resumption in Cattle     

K Gutierrez  L Siqueira  ML Rigo  WG Glanzner  JF Oliveira  PB Gonçalves 《Reproduction in domestic animals》2013,48(5):844-849

The aim of the present study was to examine the role of oxytocin (OT) in the progesterone (P4) and prostaglandins (PGs) pathway to induce oocyte meiotic resumption. Cumulus–oocyte complexes were co‐cultured with follicular hemisections for 15 h to determine the effects of different doses of OT or atosiban (ATO; oxytocin receptor antagonist) on oocyte meiotic resumption. In another experiment, we examined the effect of the interaction between P4, OT and PGs on the regulatory cascade of the oocyte meiotic resumption. Oxytocin at 1 μm was effective in inducing meiotic resumption in oocytes co‐cultured with follicular cells (84.0%), not differing from the positive control group (74.4%). Atosiban inhibited in a dose‐dependent manner the positive effect of OT on the meiotic resumption (27.6% metaphase I with 10 μm of ATO, which did not differ from the 25.5% of the negative control group). Furthermore, a third experiment showed that P4 was able to induce oocyte meiotic resumption, which was inhibited by ATO. However, the OT positive effect was not blocked by mifepristone (P4 antagonist), but was inhibited by indomethacin (a non‐selective PTGS2 inhibitor). Collectively, these data suggest a sequential role of P4, OT and PGs in the induction of oocyte meiotic resumption.  相似文献