全文获取类型
| 收费全文 | 97577篇 |
| 免费 | 4795篇 |
| 国内免费 | 32篇 |
专业分类
| 林业 | 7301篇 |
| 农学 | 3905篇 |
| 基础科学 | 636篇 |
| 13148篇 | |
| 综合类 | 12984篇 |
| 农作物 | 5116篇 |
| 水产渔业 | 6022篇 |
| 畜牧兽医 | 43730篇 |
| 园艺 | 1954篇 |
| 植物保护 | 7608篇 |
出版年
| 2018年 | 3830篇 |
| 2017年 | 3884篇 |
| 2016年 | 2491篇 |
| 2015年 | 1199篇 |
| 2014年 | 1431篇 |
| 2013年 | 3295篇 |
| 2012年 | 3295篇 |
| 2011年 | 4795篇 |
| 2010年 | 3776篇 |
| 2009年 | 2975篇 |
| 2008年 | 3997篇 |
| 2007年 | 4099篇 |
| 2006年 | 2414篇 |
| 2005年 | 2413篇 |
| 2004年 | 2233篇 |
| 2003年 | 2431篇 |
| 2002年 | 2109篇 |
| 2001年 | 2431篇 |
| 2000年 | 2422篇 |
| 1999年 | 1928篇 |
| 1998年 | 756篇 |
| 1997年 | 809篇 |
| 1996年 | 727篇 |
| 1995年 | 893篇 |
| 1994年 | 755篇 |
| 1993年 | 766篇 |
| 1992年 | 1619篇 |
| 1991年 | 1744篇 |
| 1990年 | 1700篇 |
| 1989年 | 1718篇 |
| 1988年 | 1612篇 |
| 1987年 | 1567篇 |
| 1986年 | 1641篇 |
| 1985年 | 1546篇 |
| 1984年 | 1345篇 |
| 1983年 | 1127篇 |
| 1982年 | 790篇 |
| 1979年 | 1268篇 |
| 1978年 | 975篇 |
| 1977年 | 923篇 |
| 1976年 | 850篇 |
| 1975年 | 876篇 |
| 1974年 | 1126篇 |
| 1973年 | 1136篇 |
| 1972年 | 1108篇 |
| 1971年 | 1028篇 |
| 1970年 | 952篇 |
| 1969年 | 925篇 |
| 1968年 | 781篇 |
| 1967年 | 870篇 |
排序方式： 共有10000条查询结果，搜索用时 15 毫秒
41.
42.
43.
44.
45.
Herrick GW 《Science (New York, N.Y.)》1928,68(1759):237-244
46.
47.
48.
49.
J L López-Rivero E Agüera J G Monterde J Vivo M V Rodríguez-Barbudo 《American journal of veterinary research》1992,53(5):847-850
The mean area and minimal diameter of 3 histochemically determined myofiber types (1, 2A, and 2B; myosin ATPase in acid buffer) were calculated in middle gluteal muscle biopsy specimens from 62 stallions, 47 Andalusians and 15 Arabians, ranging in age from 6 to 12 years. Fourteen Andalusians and 7 Arabians were untrained, and the remainder were actively endurance-trained. The 6-month training schedules involved walking, slow trotting, and cantering. Fourteen Andalusians were moderately endurance-trained, whereas the other 19 Andalusians and 8 Arabians were strongly endurance-trained. Significant differences were not recorded between untrained and endurance-trained Arabians with respect to the area (type 1, 3,194 +/- 869 microns 2 and 3,150 +/- 370 microns 2; type 2A, 3,819 +/- 890 microns 2 and 3,380 +/- 356 microns 2; and type 2B, 4,872 +/- 962 microns 2 and 4,417 +/- 646 microns 2) or minimal diameter (type 1, 52.2 +/- 7.4 microns and 52.8 +/- 3.1 microns; type 2A, 58.1 +/- 6.7 microns and 55.0 +/- 2.8 microns; and type 2B, 65.3 +/- 6.4 microns and 63.4 +/- 4.3 microns) of the 3 fiber types, nor between untrained and endurance-trained Andalusians with respect to the area (untrained, 3,990 +/- 690 microns 2; moderately endurance-trained, 3,882 +/- 347 microns 2; and strongly endurance-trained, 3,758 +/- 510 microns 2) and minimal diameter (untrained, 58.1 +/- 4.7 microns; moderately endurance-trained, 59.7 +/- 2.7 microns; and strongly endurance-trained, 58.7 +/- 4.5 microns) of 2A fibers.(ABSTRACT TRUNCATED AT 250 WORDS) 相似文献
50.
T. D. G. Watson † D. Gaffhey C. T. Mooney† H. Thompson‡ C. J. Packard J. Shepherd 《The Journal of small animal practice》1992,33(5):207-212
Hyperchylomicronaemia was identified in a four-week-old Siamese kitten with lethargy, in-appetence, hindlimb ataxia and profound anaemia. The kitten was euthanased and at necropsy a thrombus was found occluding the caudal aorta. Two littermates later presented with lethargy, inappetence and hypertriglyceri-daemia which resolved after being weaned on to a low fat diet. A similar condition was subsequently diagnosed in a kitten born to the same sire but a different queen. The expression of hyperchylomicronaemia in two related litters was suggestive of an inherited, familial defect in the function of lipoprotein lipase (LPL). The activity of this enzyme was reduced in all three parents, the two recovered cases and two related, but apparently unaffected kittens, compared with a control group of unrelated cats belonging to the breeder. This reduction in activity was not attributable to defective activation of LPL by its serum cofactor apolipoprotein C-II or the presence in plasma of a factor that inhibited LPL. The gene that codes for LPL was examined by restriction fragment length polymorphism analysis using a human LPL cDNA probe. The results showed that the cat has a similar, but not identical, LPL gene to man. However, there were no differences in the restriction fragment patterns obtained from affected, unaffected and control animals. 相似文献