5%锐劲特悬浮种衣剂防治黄曲条跳甲田间试验   总被引：1，自引：0，他引：1  

乔润香  张万里  程功  胡美英 《长江蔬菜》2009,(6)

5%锐劲特悬浮种衣剂防治黄曲条跳甲的田间药效试验,结果表明,每1 kg种子用5%锐劲特悬浮种衣剂100,120 mL拌种,30 d内对黄曲条跳甲防治效果良好,可以达到55.98%～87.50%,而且对小白菜具有较好的保护效果.试验期间在供试剂量下没有发现对蔬菜产生药害,可在生产上推广应用.  相似文献   

3种白蚁防治药剂对散白蚁的控制效果观察   总被引：1，自引：0，他引：1  

王思忠  谭速进  李宁  王志明  杨文锋  陈文 《农药科学与管理》2013,(12):56-58

2010年-2012年应用吡虫啉10%悬浮剂、联苯菊酯5%悬浮剂和氟虫腈0.5%粉剂等3种白蚁防治药剂对危害房屋建筑的散白蚁进行了针对性处理,对其防治效果进行了观察和统计研究。结果表明：吡虫啉10%悬浮剂、联苯菊酯5%悬浮剂、氟虫腈0.5%粉剂对房屋建筑散白蚁危害的控制率分别为：85.30%、84.01%、80.33%,具有较好的防治效果,且吡虫啉10%悬浮剂、联苯菊酯5%悬浮剂防效均显著高于氟虫腈0.5%粉剂。此外,对3种白蚁防治药剂处理后再次发生蚁害的情况进行了分析。  相似文献   

新型杀虫剂锐劲特在水稻上的残留动态研究   总被引：3，自引：0，他引：3  

周培  陆贻通  吴银良 《农业环境科学学报》2001,20(5):360-362

在河北、浙江两地同时进行了锐劲特在水稻上的残留动态试验。结果表明,锐劲特在稻田水和植株中的半衰期为3—4d,在土壤中的半衰期为15d,施用为建议剂量1倍量的25%锐劲特悬浮剂拌种,收获后糙米、稻壳中锐劲特原体及4种代谢物残留量均未超过最高残留限量。  相似文献   

水稻二化螟抗药性监测及防控对策*   总被引：3，自引：0，他引：3  

张帅  李永平  邵振润  郭井泉 《植物保护》2011,37(2):141-144

采用点滴法测定了2007-2009年采自浙江、江苏、湖北、四川等地水稻二化螟4龄幼虫对沙蚕毒素类的杀虫单、有机磷类的三唑磷、毒死蜱、苯基吡唑类的氟虫腈及微生物源的阿维菌素等杀虫剂的抗性。结果表明：上述二化螟种群对５种杀虫剂抗性分布具有明显的地区性,其中浙江、江苏大部分地区供试种群对杀虫单、三唑磷的抗性水平达40倍以上,已普遍产生高至极高水平抗性;对毒死蜱产生的抗性水平达22倍以上,已普遍产生中等水平至高水平抗性;对氟虫腈产生的抗性水平达6倍以上,已普遍产生低水平至中等水平抗性;对阿维菌素敏感至低水平抗性;湖北孝感、四川武胜种群对５种药剂尚未产生抗性或为低水平抗性。本文还对二化螟抗性防控对策进行了讨论。  相似文献   

中国水稻主产区褐飞虱对3种杀虫剂的抗性监测   总被引：5，自引：0，他引：5  

邵振润  张帅  李永平  沈晋良  龙丽萍  高聪芬  郭井泉 《农药学学报》2011,13(1):91-94

2006-2009年,用稻茎浸渍法连续监测了广西南宁市、广东阳江市、湖南东安县、福建福清市、江西上高县、湖北孝感市、浙江金华市、江苏通州市和安徽和县共9个地区褐飞虱种群对吡虫啉、噻嗪酮和氟虫腈的抗性变化。结果表明:褐飞虱种群对吡虫啉的抗性仍处于高水平至极高水平抗性阶段(105.5~459.7倍),但2009年监测到东安、孝感、上高种群对吡虫啉的抗性已有下降趋势;褐飞虱种群对氟虫腈的抗性有增长趋势,2006到2009年褐飞虱种群对氟虫腈由敏感至低水平抗性(<6.9倍)发展到了中水平至高水平抗性(13.5~43.3倍);由于2005年吡虫啉在高抗地区的禁用,褐飞虱种群对噻嗪酮的抗性上升速度加快,2009年已处于低水平至中水平抗性阶段(7.0~14.4倍)。这表明在吡虫啉、氟虫腈被禁用后,大面积单一使用噻嗪酮进行防治,褐飞虱对噻嗪酮的抗性有可能加速发展。  相似文献   

二化螟对氟虫腈抗性初探   总被引：9，自引：3，他引：9  

姜卫华  韩召军  郝鸣丽 《中国水稻科学》2005,19(6):577-579

 采用点滴测定法，于2001~2004年对江、浙、皖3省12个县市的二化螟进行了氟虫腈的抗性调查。江苏及安徽等地二化螟对氟虫腈仍处于敏感状态，但江苏无锡和浙江慈溪二化螟的敏感性明显下降（抗性倍数RR分别为3.1和3.6倍），浙江苍南二化螟已出现中等水平的抗性（RR为21.2倍）。由此认为，二化螟目前仍处于对氟虫腈抗性发展的早期阶段。解毒酶抑制剂的活体增效实验发现，TPP、DEM能明显提高氟虫腈对抗性二化螟的毒力（增效比SR分别为1.8和1.6），但PBO在敏感性和抗性二化螟中的增效作用均不显著（SR为1.1~1.2）。表明酯酶和谷胱甘肽转移酶与二化螟对氟虫腈抗性的形成有一定关系。不同杀虫剂的比较毒力测定表明，三唑磷和敌百虫由于产生了抗性，对二化螟的毒力很低，乙酰甲胺磷本身对二化螟的毒力也不高，已很难再用来防治抗性二化螟。但哒嗪硫磷、二嗪磷、溴氰菊酯和阿维菌素对二化螟的毒力较高，而且尚没有明显的抗性产生，在二化螟抗性治理中，可以用作氟虫腈的轮换防治药剂。  相似文献   

Toxicity and horizontal transfer of chlorantraniliprole against the Asian subterranean termite Coptotermes gestroi (Wasmann): effects of donor:recipient ratio, exposure duration and soil type     

Neoh KB  Hu J  Yeoh BH  Lee CY 《Pest management science》2012,68(5):749-756

BACKGROUND: The effectiveness of chlorantraniliprole and other insecticides (bifenthrin, fipronil, indoxacarb, imidacloprid and chlorfenapyr) were tested against Coptotermes gestroi (Wasmann). Four experiments were conducted: a topical bioassay, a horizontal transfer study, an insecticide bioavailability test and a feeding bioassay. RESULTS: The topical bioassay showed that chlorantraniliprole was significantly less active to C. gestroi at 24 h post‐treatment compared with the other insecticides tested. Nevertheless, it is likely that a lesser amount of chlorantraniliprole was required to cause 50% mortality of C. gestroi at 7 and 14 days post‐treatment. The exposure duration and donor:recipient ratio affect the mortality of recipient termites. Mortality after exposure to chlorantraniliprole in sandy clay was significantly lower than in sand; however, by 14 days, > 90% of donor and recipient termites died in both substrates, irrespective of concentration. Fipronil and imidacloprid showed faster action, and high to moderate toxicity to C. gestroi. Termite workers also ceased to feed after exposure for 1 h to 50 mg kg^?1 chlorantraniliprole‐treated sandy clay. CONCLUSION: Chlorantraniliprole demonstrated delayed toxicity at the lowest label rate (50 mg kg^?1) in sandy clay. Its slow action will enable greater transfer of toxicant between nestmates, while feeding cessation will promote greater social interaction between healthy and exposed termites. Copyright © 2011 Society of Chemical Industry  相似文献   

Identification of the Rdl mutation in laboratory and field strains of the cat flea, Ctenocephalides felis (Siphonaptera: Pulicidae)     

Bass C  Schroeder I  Turberg A  Field LM  Williamson MS 《Pest management science》2004,60(12):1157-1162

In many insect species, resistance to cyclodiene insecticides is caused by amino acid substitutions at a single residue (A302) within the M2 transmembrane region of the gamma-aminobutyric acid (GABA) receptor sub-unit termed Rdl (resistance to dieldrin). These mutations (A302S and A302G) have also been shown to confer varying levels of cross-resistance to fipronil, a phenylpyrazole insecticide with a similar mode of action to cyclodienes. To investigate the possible occurrence of these mutations in the cat flea, Ctenocephalides felis (Bouché), a 176-bp fragment of the cat flea Rdl gene, encompassing the mutation site, was PCR amplified and sequenced from nine laboratory flea strains. The A302S mutation was found in eight of the nine strains analysed, although the relative frequency of the mutant allele varied between strains. Only one strain (R6) was found to be homozygous for the S302 allele in all the individuals tested, and this correlated with previous reports of low-level fipronil resistance in this strain. A PCR-based diagnostic assay, capable of screening individual fleas for this mutation, was developed and used to survey a range of fleas collected at random from veterinary clinics in the UK and USA. The A302S mutation was present at a high frequency in these domestic pet populations.  相似文献   

Genetics of spinosad resistance in a multi-resistant field-selected population of Plutella xylostella   总被引：2，自引：0，他引：2  

Sayyed AH  Omar D  Wright DJ 《Pest management science》2004,60(8):827-832

Resistance to the bacteria-derived insecticides spinosad (Conserve), abamectin (Vertimec), Bacillus thuringiensis var kurstaki (Btk) (Dipel), B thuringiensis var aizawai (Bta) (Xentari), B thuringiensis crystal endotoxins Cry1Ac and Cry1Ca, and to the synthetic insecticide fipronil was estimated in a freshly-collected field population (CH1 strain) of Plutella xylostella (L) from the Cameron Highlands, Malaysia. Laboratory bioassays at G1 indicated significant levels of resistance to spinosad, abamectin, Cry1Ac, Btk, Cry1Ca, fipronil and Bta when compared with a laboratory insecticide-susceptible population. Logit regression analysis of F1 reciprocal crosses indicated that resistance to spinosad in the CH1 population was inherited as a co-dominant trait. At the highest dose of spinosad tested, resistance was close to completely recessive, while at the lowest dose it was incompletely dominant. A direct test of monogenic inheritance based on a back-cross of F1 progeny with CH1 suggested that resistance to spinosad was controlled by a single locus.  相似文献   

Cross-resistance potential of fipronil in Musca domestica   总被引：4，自引：0，他引：4  

Kristensen M  Jespersen JB  Knorr M 《Pest management science》2004,60(9):894-900

The toxicity of fipronil to insecticide-susceptible houseflies and the cross-resistance potential of fipronil were determined for six insecticide-resistant laboratory housefly strains by topical application and feeding bioassay. The insecticide-resistant strains represented different levels and patterns of resistance to pyrethroids, organophosphates, carbamates and organochlorines. Five strains were almost susceptible to fipronil in feeding bioassay with resistance factors at LC50 between 0.36 and 3.0. Four of these strains were almost susceptible to topically applied fipronil (resistance factors at LD50 were 0.55, 0.83, 3.3 and 2.5, respectively), whereas one strain was 13-fold resistant to topically applied fipronil. A highly gamma-HCH-resistant strain, 17e, was 430-fold resistant to fipronil in topical application bioassay and 23-fold resistant in feeding bioassay at LD50/LC50. We also tested the toxicity of fipronil in a feeding bioassay and gamma-HCH in topical application bioassay on thirteen housefly field populations. Eleven of the field populations had resistance factors for fipronil ranging from 0.98 to 2.4 at LC50, whereas two populations were 4.0- and 4.6-fold resistant to fipronil. The resistance level to gamma-HCH at LD50 in the field populations ranged from 1.8- to 8.1-fold. The two strains showing fipronil resistance were 3.4- and 8.1-fold resistant to gamma-HCH. Fipronil and gamma-HCH toxicities were positively correlated in the field populations. Biochemical assays of esterase, glutathione S-transferase and cytochrome P450 monooxygenase indicated that the low fipronil resistance observed in laboratory and field strains could be caused by elevated detoxification or be due to a target-site resistance mechanism with cross-resistance to gamma-HCH.  相似文献