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Giannina Vizzotto Ivica Matosevic Roberto Pinton Zeno Varanini Guglielmo Costa 《Journal of plant nutrition》2013,36(2-3):327-334
Many dicotyledonous species respond to iron (Fe) deficiency by morphological and physiological changes at root level, which are usually defined as Strategy I. Particularly, these latter modifications include a higher acidification of the external medium and the induction of a high root Fe reductase activity. The aim of this work was to investigate the response of kiwi (Actinidia deliciosa cv. Hayward) plants, which often exhibit Fe chlorosis in the field, to Fe deficiency. Actinidia kept for two weeks in nutrient solution without Fe showed visual deficiency symptoms (leaf chlorosis). Moreover, upon prolonged micronutrient shortage shoot, and to a lesser extent, root dry weight accumulation was greatly impaired. Roots of Fe‐deficient Actinidia showed an increased capacity of net proton extrusion and higher ferric ethylenediaminetetraacetate [Fe(III)EDTA] reductase activity as compared to plants grown in the presence of 10 μM Fe(III)EDTA. Localization of the increased acidification and reductase capacity by means of agar‐technique revealed that these activities are both present in the sub‐apical region of the roots. Re‐supply of Fe after two weeks partially reversed the tendency of the roots to acidify the nutrient solution and to reduce Fe(III)EDTA. 相似文献
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Anna Giannina Perugini Maria Rosaria Carullo Assunta Esposito Vincenzo Caligiuri Federico Capuano Giorgio Galiero Giuseppe Iovane 《Veterinary research communications》2010,34(4):387-392
In the last two decades, the emergence and spread of antimicrobial-resistant pathogens, among them Salmonella, has become
a serious health hazard worldwide, and specifically the high incidence of multidrug resistance has been encountered widely
in many European countries. This study examines the antimicrobial supsceptibility of Salmonella enterica strains Typhimurium
and Enteritidis isolated in Campania and Calabria region (Southern Italy) from animal and food of animal origin. The relationship
of antibiotic resistance phenotype and the presence of some resistance genes has been also investigated. As espected, our
results showes that resistance to ampicillin, chloramphenicol, streptomycin, sulphonamides and tetracycline is common, although
resistance to other antibiotics (i.e.:nalidixic acid) and other resistance patterns occur. The genetic resistant patterns
have been partially described for this food-borne pathogen but efforts are needed to realize the complete characterization
of antimicrobial resistance genes. 相似文献
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Alleviating neuropathic pain hypersensitivity by inhibiting PKMzeta in the anterior cingulate cortex
Li XY Ko HG Chen T Descalzi G Koga K Wang H Kim SS Shang Y Kwak C Park SW Shim J Lee K Collingridge GL Kaang BK Zhuo M 《Science (New York, N.Y.)》2010,330(6009):1400-1404
Synaptic plasticity is a key mechanism for chronic pain. It occurs at different levels of the central nervous system, including spinal cord and cortex. Studies have mainly focused on signaling proteins that trigger these plastic changes, whereas few have addressed the maintenance of plastic changes related to chronic pain. We found that protein kinase M zeta (PKMζ) maintains pain-induced persistent changes in the mouse anterior cingulate cortex (ACC). Peripheral nerve injury caused activation of PKMζ in the ACC, and inhibiting PKMζ by a selective inhibitor, ζ-pseudosubstrate inhibitory peptide (ZIP), erased synaptic potentiation. Microinjection of ZIP into the ACC blocked behavioral sensitization. These results suggest that PKMζ in the ACC acts to maintain neuropathic pain. PKMζ could thus be a new therapeutic target for treating chronic pain. 相似文献
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