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Evoked potentials were produced by anodal stimulation over the motor cortex in six dogs. Potentials were recovered from the cranial thoracic and caudal lumbar portions of the spinal cord, and the radial and sciatic nerves. Evoked potential averages were recorded every 1.5 minutes during 40 minutes of aortic occlusion and during 40 minutes of reperfusion. Mean amplitudes of evoked potentials recovered from the caudal lumbar spinal cord decreased to 50% of original values at minute 12.2. Upon release of occlusion, the evoked potentials returned to baseline levels and remained there throughout the period of reperfusion. Sciatic nerve amplitudes decreased to 50% of original values at minute 4.5. In no subject could wave forms be recovered after minute 9.0. Upon release of occlusion, the evoked potentials returned to baseline levels and above, then deteriorated to 29 +/- 12% after 40 minutes of reperfusion. We concluded that transcranially induced evoked potentials were highly sensitive to spinal cord ischemia. Evoked potentials recovered from the sciatic nerve were consistent with functional grey matter immediately upon reperfusion, but deteriorated during reperfusion.  相似文献   
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Urethrotomy was performed on 12 normal male dogs at the base of the os penis. The surgical site was allowed to heal by second intention in six dogs, and suture closure of the urethral mucosa, tunica albuginea, and subcuticular tissue was performed in six dogs. All dogs were maintained for 60 days postoperatively. Packed cell volume and serum total protein were measured before surgery and at 2, 4, 7, 14, and 60 days postoperatively. Suture closure of the urethra resulted in less hemorrhage during the postoperative recovery period. Urethral mucosal irregularity was seen in urethrograms performed on all operated dogs 60 days postoperatively. Urethral stricture was not observed in any of the dogs in this study.  相似文献   
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