| 植物Cu,Zn-SOD分子调控机理研究进展 |
| |
| 引用本文： | 姚 婕,赵艳玲.植物Cu,Zn-SOD分子调控机理研究进展[J].热带作物学报,2014,34(3):55-60. |
| |
| 作者姓名： | 姚 婕 赵艳玲 |
| |
| 作者单位： | 华侨大学化工学院 福建厦门 361021;华侨大学化工学院 福建厦门 361021 |
| |
| 摘 要： | 铜/锌超氧化物歧化酶(Cu,Zn-SOD)能清除植物体内有害的活性氧(ROS)，参与植株遭受逆境胁迫时的应激反应等过程。铜分子伴侣(CCS)可以传递铜离子到Cu,Zn-SOD当中，并将其激活生成有活性的酶分子，依赖CCS协助是Cu,Zn-SOD主要的激活途径。植物在铜缺乏的环境下会诱导启动子结合蛋白(SPL7)和小RNA398(miR398)的表达，miR398通过降解编码Cu,Zn-SOD的mRNA抑制Cu,Zn-SOD的生成，从而调控植物体内铜平衡。本文主要对植物Cu,Zn-SOD激活和调控途径进行综
|
| 关 键 词： | Cu Zn-SOD CCS miR398 SPL7 调控途径 |
Molecular Regulation Mechanism Research Progress on Plant Cu,Zn-SOD |
| |
| Institution: | College of Chemical Engineering, Huaqiao University, Xiamen, Fujian 361021;College of Chemical Engineering, Huaqiao University, Xiamen, Fujian 361021 |
| |
| Abstract: | Superoxide dismutases (Cu,Zn-SODs) are important antioxidant enzymes that catalyze the disproportionation of superoxide anion to oxygen and hydrogen peroxide to guard cells against superoxide toxicity. The major pathway for activation of copper/zinc SOD (CSD) requiring the CCS copper chaperone to insert copper and activate SOD1 through oxidation of an intramolecular disulfide. Expression of miR398 and SPL7 (for SQUAMOSA promoter binding protein-like7) are induced in response to copper deficiency and miR398 is involved in the degradation of mRNAs encoding copper/zinc superoxide dismutase. This paper reviewed on plant Cu, Zn-SOD activation and regulatory pathways. |
| |
| Keywords: | Cu Zn-SOD CCS miR398 SPL7 regulatory pathways |
|
| 点击此处可从《热带作物学报》浏览原始摘要信息 |
| 点击此处可从《热带作物学报》下载免费的PDF全文 |
|
