共查询到20条相似文献，搜索用时 328 毫秒
1.
猪附红细胞体病是一种由猪附红细胞体寄生于血液中的原虫病，临床上以发热、皮肤发红、苍白、消瘦等为主要特征。近几年来，猪附红细胞体病的发生已缺乏明显的季节性，常呈零星发生或隐性感染，受应激或混合感染时发病。该文介绍了一例猪附红细胞体病的流行情况，通过临床诊断和采取综合防治措施，取得显著疗效。 相似文献
2.
本文结合本地一规模化猪场暴发猪附红细胞体病的控制经验，从病原学牲、致病机理、流行特点、临床症状、病理变化、诊断、治疗等方面认识猪附红细胞体病，探讨综合防制措施。 相似文献
3.
张来东 《畜牧兽医科技信息》2015,(3):100-101
猪附红细胞体病是由猪附红细胞体引发的传染病,黄疸与贫血是其主要特征。本文探讨了猪附红细胞体病的流行特点和临床症状,介绍了几种有效的治疗方法,并提出了预防猪附红细胞体病的措施。 相似文献
4.
猪附红细胞体病的特征性症状１９９５年７月１４日，我场流行了一场猪附红细胞体病。在发病的过程中发现一些症状是属于该病的特征性症状。了解猪附红细胞体病的特有症状，有助于我们对该病及早做出准确的诊断。猪附红细胞体病流行一开始．少数猪或个别猪食欲不振，精神萎... 相似文献
5.
猪附红细胞体病是由猪附红细胞体寄生于红细胞表面或血浆中引起严重危害养猪生产的一种疾病。2003年以来宁德市部分县(乡)发生猪附红细胞体病。笔者遇到一起猪病,通过流行病学调查、临床 相似文献
6.
猪附红细胞体病是由许多因素引起的,尤其在受到强烈因素如突然更换圈舍、饲料,天气恶劣,环境卫生条件差等影响,就可能诱发猪附红细胞体病的发生。夏季是猪附红细胞体病的高发季节,临床上必须采取综合措施进行防治。1流行季节本病多发于夏秋雨水较多吸血昆虫活动频繁的高峰 相似文献
7.
附红细胞体病是由附红细胞体寄生于人畜红细胞表面、血浆及骨髓所引起的一种人畜共患传染病。根据猪附红细胞体病的流行特征、传播途径,阐述了猪附红细胞体病临床症状以及病理剖检和细胞形态学变化,并提出预防和治疗该病的措施。 相似文献
8.
9.
附红细胞体病是由附红细胞体寄生于人和动物红细胞表面或游离在血浆中引起的一种以贫血、黄疸、发热等为主要临床特征的人畜共患病。近年来，我国河北、天津、山西等省市猪附红细胞体病的发生和流行呈明显的上升趋势，尤其是对仔猪的危害更为严重。2002年8月初，四川省成都市某养殖场暴发猪附红细胞体病，现报告如下。 相似文献
10.
11.
Fukai K Morioka K Yoshida K 《The Journal of veterinary medical science / the Japanese Society of Veterinary Science》2011,73(9):1207-1210
In this study, we carried out an experimental infection in pigs using a foot-and-mouth disease virus isolated from the 2010 epidemic in Japan to analyze the clinical manifestation, antibody response and virus shedding patterns in pigs. We found that the virus was virulent in pigs, producing a synchronous disease in the inoculated pigs and efficient spread to direct contact pigs. These results are useful for epidemiologically investigating the 2010 epidemic in Japan and improving the measures for controlling possible future FMD outbreaks in Japan or elsewhere. 相似文献
12.
Stegeman A Elbers AR Bouma A de Smit H de Jong MC 《Preventive veterinary medicine》1999,42(3-4):201-218
In this paper, we describe the transmission of Classical Swine Fever virus (CSF virus) within herds during the 1997–1998 epidemic in the Netherlands. In seven herds where the infection started among individually housed breeding stock, all breeding pigs had been tested for antibodies to CSF virus shortly before depopulation. Based upon these data, the transmission of CSF virus between pigs was described as exponential growth in time with a parameter r, that was estimated at 0.108 (95% confidence interval (95% CI) 0.060–0.156). The accompanying per-generation transmission (expressed as the basic reproduction ratio, R0) was estimated at 2.9. Based upon this characterisation, a calculation method was derived with which serological findings at depopulation can be used to calculate the period in which the virus was with a certain probability introduced into that breeding stock. This model was used to estimate the period when the virus had been introduced into 34 herds where the infection started in the breeding section. Of these herds, only a single contact with a herd previously infected had been traced. However, in contrast with the seven previously mentioned herds, only a sample of the breeding pigs had been tested before depopulation (as was the common procedure during the epidemic). The observed number of days between the single contact with an infected herd and the day of sampling of these 34 herds fitted well in the model. Thus, we concluded that the model and transmission parameter was in agreement with the transmission between breeding pigs in these herds.
Because of the limited sample size and because it was usually unknown in which specific pen the infection started, we were unable to estimate transmission parameters for weaned piglets and finishing pigs from the data collected during the epidemic. However, from the results of controlled experiments in which R0 was estimated as 81 between weaned piglets and 14 between heavy finishing pigs (Laevens et al., 1998a. Vet. Quart. 20, 41–45; Laevens et al., 1999. Ph.D. Thesis), we constructed a simple model to describe the transmission of CSF virus in compartments (rooms) housing finishing pigs and weaned piglets. From the number of pens per compartment, the number of pigs per pen, the numbers of pigs tested for antibodies to CSF virus and the distribution of the seropositive pigs in the compartment, this model gives again a period in which the virus most probably entered the herd. Using the findings in 41 herds where the infection started in the section of the finishers or weaned piglets of the age of 8 weeks or older, and of which only a single contact with a herd previously infected was known, there was no reason to reject the model. Thus, we concluded that the transmission between weaned piglets and finishing pigs during the epidemic was not significantly different from the transmission observed in the experiments. 相似文献
13.
14.
姜雨君 《畜牧兽医科学(电子版)》2021,(5):105-106
猪皮炎肾病综合症是由圆环病毒二型感染引起的一种以猪皮肤表面出现红疹、丘疹、结痂或坏死,肾脏严重肿大为主要临床特征的常见传染性疾病。皮炎肾病综合症主要危害生长猪和育肥猪。受病毒感染后,患病猪表现为生长发育不良,免疫功能受到严重抑制,易继发感染其他传染性疾病,表现出复杂的临床症状,给生猪养殖业造成严重损失和危害。防范疾病的发生流行,需要掌握猪皮炎肾病综合症的具体流行特点、临床表现,并采取针对有效措施进行诊断,然后构建有效的防控措施,短时间内控制病情,避免损失进一步加大。该文主要论述猪皮炎肾病综合症的流行特点、临床症状、病理变化、诊断方法和防治措施。 相似文献
15.
16.
伪狂犬病病毒（PRV）是α疱疹病毒属的一个成员，为伪狂犬病的病原，猪是伪狂犬病病毒的自然宿主，该病原一旦感染猪，则产生潜伏感染，其致死率随猪年龄的增加而降低。由于伪狂犬病的暴发引起畜牧业的巨大经济损失，许多国家都进行免疫接种预防该病，而该病在不同的国家地区危害不同，所造成的经济损失也不一样，另外各国的经济发展不平衡，因此对该病所采取的防制策略亦有所不同，文章综述了美国，日本，欧共体及其成员国之间对伪狂犬病采取的防制策略及取得的进展，并根据我国伪狂犬病的流行现状，提出了应采取的控制策略。 相似文献
17.
18.
为了探究福建省龙岩地区的2018年猪伪狂犬病野毒感染及流行概况,本试验随机选取龙岩新罗区、连城县、武平县以及长汀县的部分规模化猪场共21个,应用ELISA法对763份血清样品PRV g E抗体进行血清学调查。结果显示样品检测总阳性率为30.41%,其中保育猪、育肥猪、能繁母猪及后备母猪的阳性率分别为:25.29%、30.98%、30.85%、31.75%;用SPSS19.0对其进行差异显著性分析,结果显示不同阶段的猪群gE抗体阳性率差异不显著(P=0.741>0.05);各场场均阳性率为31.89%;各地区阳性率分别为:41.58%、18.18%、30.14%、26.29%,场均阳性率分别为41.14%、23.50%、29.60%、33.33%,对其进行差异显著性分析,结果表示不同地区之间gE抗体阳性率差异极显著(P=0.0003<0.001),即各地区之间伪狂犬病野毒感染情况差异较大,部分地区更需要加强其控制和净化措施。 相似文献
19.
Stegeman A Elbers A de Smit H Moser H Smak J Pluimers F 《Veterinary microbiology》2000,73(2-3):183-196
In 1997, the pig husbandry in the Netherlands was struck by a severe epidemic of classical swine fever (CSF). During this epidemic 429 CSF-infected herds were depopulated and approximately 1300 herds were slaughtered pre-emptively. In addition millions of pigs of herds not CSF-infected were killed for welfare reasons (over crowding or overweight). In this paper, we describe the course of the epidemic and the measures that were taken to control it.The first outbreak was detected on 4 February 1997 in the pig dense south-eastern part of the Netherlands. We estimate that CSF virus (CSFV) had already been present in the country by that time for 5-7 weeks and that the virus had been introduced into approximately 39 herds before the eradication campaign started. This campaign consisted of stamping-out infected herds, movement restrictions and efforts to diagnose infected herds as soon as possible. However, despite these measures the rate at which new outbreaks were detected continued to rise. The epidemic faded out only upon the implementation of additional measures such as rapid pre-emptive slaughter of herds in contact with or located near infected herds, increased hygienic measures, biweekly screening of all herds by veterinary practitioners, and reduction of the transportation movements for welfare reasons. The last infected herd was depopulated on 6 March 1998. 相似文献