共查询到20条相似文献，搜索用时 312 毫秒
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不同品种嫩果椰水主要品质性状、矿质元素含量分析 总被引：2，自引：0，他引：2
以海南4个主栽椰子品种为试材,研究不同品种嫩果椰水基本性状间及椰水中的矿质元素(K、Ca、Na、Mg、Fe、Mn、Cu、Zn)含量间的差异。结果表明,嫩果椰子水的pH平均值为5.50～5.58;可溶性固形物含量为5.60%～7.75%;测定的8种矿质营养元素中,K含量最高,达355.93mg/kg,Na为32.70mg/kg,Ca为6.36mg/kg,Mn为2.67mg/kg。各品种间存在显著和极显著差异。3个矮种椰子品种均具有各自特点,均可作为嫩果椰子品种发展,可根据不同特色矮种椰子的观赏价值、生态价值、经济价值和营养价值,规模栽培矮种椰子,发展海南热带旅游观光农业。 相似文献
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菲律宾椰子协会联台会公布的最新统计数据,今年头两个月,菲律宾椰产品出口量约为29.3万吨,比去年同期下降了17％。 菲椰子协会联合会认为,今年头两个月菲椰产品出口下降主要是由于椰油和于椰肉出口大幅减少造成的。今年头两个月,椰油占椰产品 相似文献
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椰心叶甲Brontispa longissima（Gestro）是棕榈科植物的一种毁灭性害虫，属鞘翅目Coleoptera，叶甲总科Chrysomeloidea，铁甲科Hispidae，潜甲亚科Anisoderiane，平胸族Cryptonychini。2002年6月，首次在海南省发现椰心叶甲后，短短两三年内广东、海南等地的棕榈科植物受到了严重危害。椰心叶甲在中国境内属外来入侵物种，缺少天敌，造成椰心叶甲在国内为害猖獗，在海南省的18个县市中就有12个县市发生椰心叶甲危害。 相似文献
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明椒15号是以m79-4-1-2-3为母本、f69-2-3-5-4为父本选育而成的早熟薄皮辣椒杂交1代品种。该品种早熟,门椒节位6~8节,从定植至红椒始收55 d左右,全生育期160 d左右,果长18~20 cm,横径3.0~3.5 cm,果肉厚度0.2 cm,单果质量50~60 g,667 m2产量2200~2500 kg,青熟果绿色,老熟果红色,果皮微皱、辣度值(SHU,Scoville heat units,斯科维尔指数)为6652,维生素C质量分数157 mg·100 g-1,粗纤维质量分数0.04%,香味浓郁,品质佳,抗病、抗逆性较强,适宜福建省内作春秋保护地栽培。2020年通过农业农村部非主要农作物品种登记。 相似文献
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AIM: To investigate the role of microRNA-101-3p (miRNA-101-3p) on the proliferation, apoptosis and invasion of gastric cancer cells and the possible regulatory mechanisms. METHODS: The expression of miRNA-101-3p in two kinds of gastric cancer cells and a gastric mucosal cell line was detected by real-time PCR. The miRNA-101-3p was overexpressed by Lipofectamine 2000 transfection with miRNA-101-3p mimics. The effects of miRNA-101-3p on cell cycle distribution and apoptosis were analyzed by flow cytometry. The effects of miRNA-101-3p on cell proliferation and migration abilities were detected by CCK-8 assay, trypan blue exclusion test and Transwell assay. The protein expression of enhancer of zeste homolog 2 (EZH2) was determined by Western blot. RESULTS: The expression of miRNA-101-3p in gastric cancer cells was lower than that in gastric mucosal cells (P<0.05). The gastric cancer cell MGC-803 had the lowest expression level of miRNA-101-3p. The result of flow cytometry showed that the population of S phase was reduced, and the population of G0/G1 phase and the early stage apoptotic rate were increased after the expression of miRNA-101-3p was overexpressed (P<0.05). The results of CCK-8 assay, trypan blue exclusion test and Transwell assay showed that overexpression of miRNA-101-3p significantly reduced the proliferation and migration abilities of gastric cancer cells (P<0.05). Overexpression of miRNA-101-3p decreased the protein level of EZH2 (P<0.05). CONCLUSION: miRNA-101-3p may suppresses the gastric cancer cell proliferation and migration, and promotes the gastric cancer cell apotosis by down-regulation of EZH2. 相似文献
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AIM:To investigate the expression and clinical significance of microRNA-139-3p (miR-139-3p) in the apoptosis model of cardiomyocytes induced by hypoxia. METHODS:Under normal and hypoxic conditions, the expression of miR-139-3p in neonatal rat cardiomyocytes was detected by RT-qPCR. miR-139-3p inhibitor and miR-139-3p inhibitor negative control were transfected into the primary neonatal rat cardiomyocytes. The transfected cardiomyocytes were cultured in closed anoxic box (95% N2 and 5% CO2) at 37℃ for 12 h. Flow cytometry and Western blot were used to determine the apoptosis of cardiomyocytes. RESULTS:After hypoxia for 12 h, the expression level of miR-139-3p and the apoptotic rate of the cardiomyocytes in hypoxia group were significantly higher than those in normal group (P<0.05). Moreover, compared with the miR-139-3p inhibitor negative control group, the apoptotic rate of the cardiomyocytes was significantly decreased in miR-139-3p inhibitor group (P<0.05). CONCLUSION:The expression of miR-139-3p is signi-ficantly increased in apoptotic neonatal rat cardiomyocytes induced by hypoxia. Inhibition of miR-139-3p expression reduces hypoxia-induced apoptosis of cardiomyocytes. 相似文献
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HU Ming LI Jiao LIU Ning-ning HUANG Zhen-jun WU Chong-hai ZHONG Yun LIU Shi-ming 《园艺学报》2015,31(3):524-529
AIM: To investigate the role of microRNA-486-5p (miR-486-5p) in the apoptosis of human bone marrow mesenchymal stem cells (hMSCs) induced by hydrogen peroxide (H2O2). METHODS: The hMSCs were cultured in vitro and exposed to serum-free medium and H2O2 (10 mmol/L). The changes of miR-486-5p expression in oxidative stress-related apoptosis of hMSCs were measured by real-time PCR. The hMSCs were transfected with miR-486-5p mimic or inhibitor at concentration of 30 nmol/L by Lipofectamine RNAiMAX. The effect of miR-486-5p on H2O2-induced decrease in cell viability was evaluated by MTT assay. Hoechst 33342 staining and flow cytometry were applied to determine the role of miR-486-5p in the apoptosis of hMSCs. The protein expression was evaluated by Western blotting. Caspase-3 activity was determined using a caspase-3 activity kit. RESULTS: Compared with control group, the expression of miR-486-5p significantly decreased after treated with H2O2 (P<0.05). In addition, over-expression of miR-486-5p in the hMSCs reduced the cell viability, accelerated apoptosis, down-regulated Bcl-2/Bax ratio, caspase-3 enzyme precursor content and phosphorylation of Akt, and activated caspase-3 activity. Conversely, down-regulation of miR-486-5p significantly inhibited H2O2-induced cell apoptosis and the caspase-3 activity, increased cell viability and up-regulated Bcl-2/Bax ratio and phosphorylation level of Akt. CONCLUSION: Over-expression of miR-486-5p promotes H2O2-induced hMSCs apoptosis, and repression of miR-486-5p protects hMSCs from H2O2-induced cellular apoptosis, which may be mediated by regulating Akt signaling pathway. 相似文献
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LIU Hua ZHANG Su-lan LIANG Tian-song WANG Juan ZHAO Jing-yi ZHENG Ying-juan ZHANG Xiang-xian YANG Dao-ke 《园艺学报》2000,36(10):1789-1795
AIM To investigate the expression level of long noncoding RNA （lncRNA） TTN antisense RNA 1 （TTN-AS1） in lung adenocarcinoma tissues and the effects of TTN-AS1 silencing on the viability and invasion of lung adenocarcinoma A549 cells. METHODS RT-qPCR was used to detect the expression of TTN-AS1， microRNA-519d-3p （miR-519d-3p） and matrix metalloproteinase 2 （MMP2） mRNA in 32 cases of lung adenocarcinoma and adjacent normal tissues. The untransfected A549 cells were divided into blank group， si-NC group （with si-NC transfection） and si-lncRNA group （with silencing of lncRNA TTN-AS1 expression）， with n =5 in each group. The effects of TTN-AS1 silencing on the viability and invasion of A549 cells were detected by CCK8 and Transwell methods. The targeting regulatory effects of TTN-AS1 on miR-519d-3p and miR-519d-3p on MMP2 were determined by dual-luciferase reporter assay， RNA immunoprecipitation test， RT-qPCR and Western blot. RESULTS The expression level of TTN-AS1 in 32 cases of lung adenocarcinoma tissues is notably higher than that in the adjacent normal tissues （P <0.05）. Silencing of TTN-AS1 in A549 cells significantly suppressed the cell viability and invasion. TTN-AS1 negatively regulated the expression of miR-519d-3p via sponging and absorbing miR-519d-3p. MMP2 is the target gene of miR-519d-3p and can be negatively regulated by miR-519d-3p. Overexpression of MMP2 partially reversed the inhibitory effect of TTN-AS1 silencing and miR-519d-3p overexpression on the invasion of A549 cells. CONCLUSION The lncRNA TTN-AS1 is overexpressed in lung adenocarcinoma tissues， and it regulates lung adenocarcinoma A549 cell viability and invasion via miR-519d-3p/MMP2 pathway. 相似文献